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Oxymetholone, sold under the brand names Anadrol and Anapolon among others, is an androgen and anabolic steroid (AAS) medication which is used primarily in the treatment of anemiaand type 2 diabetes. AASs have also been associated with cancer development in animal and human studies.1–35 The present paper reviews all major and minor studies on anabolic-androgenic steroids from 1977 to 2006. These data were analyzed using descriptive statistics and meta-regression techniques. In total, 19 studies investigated anabolic steroids and breast cancer incidence, and 17 studies investigated anabolic steroids and prostate cancer incidence. In 4 studies, anabolic steroids did not significantly increase the incidence of breast cancer.1 , 13 , 14 , 21 , 20 , 25 , 32 , 35 In 7 studies, anabolic steroids increased breast cancer risk by 6% compared with testosterone or human chorionic gonadotropin, but these results are based only on one study, 17 so the magnitude of this apparent increased breast cancer risk cannot be verified. A total of 9 studies reported increased risk of breast cancer associated with androgens, including: testosterone, 17 , 25 , 32 , 35 the synthetic forms of testosterone and human chorionic gonadotropin, 34 androgens like estradiol, 17 , 20 , 25 , 28 , 31 , 36 androgens like testosterone, 17 , 25 , 32 , 35 and the synthetic forms of testosterone and human chorionic gonadotropin, 34 androgens like estradiol, 17 , 20 , 25 , 32 , 35 , 36 3 , 5 , 9 , 16 , 24 , 47 , 50 . One study reported decreased risk of breast cancer associated with androgens compared with estradiol, 17 but this study is based on a population of men who participated in an exercise study with exercise in the form of high-intensity interval training, and thus the study design is not applicable to normal populations. However, some of the reported anabolic androgenic steroid associations with breast cancer risk in humans appear not to be true. For example, one long-term study found an increased risk of breast cancer with androgens compared with estradiol, 17 but the study was limited to women in the 50 to 74 year age range at the time of diagnosis, and thus the observed anabolic-androgenic steroid association is not applicable to normal populations or to any population. A study on the hormonal mechanism of estrogen-induced carcinogenesis found that high doses of androgens induce malignant transformation in certain cell types. 17 The mechanism is believed to involve inhibition of estrogen receptor-mediated mitogenic and angiogenic events. 17 , 20 Another study suggested the potential of the androgen receptor to influence cancer Related Article: